FAK-话题@MedSci

#FAK#

18篇内容 | 1107人围观

关注话题

话题活跃用户

#插入话题

插入图片

最新最热

[GetTopicListResponse(id=1640590, encodeId=b4b816405906a, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=ASCO 2022 | FAKTION临床：capivasertib+氟维司群，改善芳香化酶抑制剂耐药MBC患者生存！, objectType=article, longId=729099, objectId=c01ae29099c1, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=c01ae29099c1, replyNumber=0, likeNumber=83, createdTime=2022-06-14, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=c01ae29099c1, moduleTitle=ASCO 2022 | FAKTION临床：capivasertib+氟维司群，改善芳香化酶抑制剂耐药MBC患者生存！, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=c01ae29099c1)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640589, encodeId=dbb216405897c, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Diabetologia：IGFBP-1在人2型糖尿病肾小球中的表达降低并调节人足细胞中的β1-整联蛋白/FAK信号传导, objectType=article, longId=217988, objectId=d47021e988e9, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=d47021e988e9, replyNumber=0, likeNumber=78, createdTime=2021-11-21, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=d47021e988e9, moduleTitle=Diabetologia：IGFBP-1在人2型糖尿病肾小球中的表达降低并调节人足细胞中的β1-整联蛋白/FAK信号传导, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=d47021e988e9)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640588, encodeId=9ce51640588ef, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Artif Cells Nanomed Biotechnol：FAK在滑膜间充质干细胞中通过Wnt和MAPK信号通路介导BMP9诱导的成骨分化, objectType=article, longId=174583, objectId=e2b61e4583cc, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=e2b61e4583cc, replyNumber=0, likeNumber=69, createdTime=2020-06-07, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=e2b61e4583cc, moduleTitle=Artif Cells Nanomed Biotechnol：FAK在滑膜间充质干细胞中通过Wnt和MAPK信号通路介导BMP9诱导的成骨分化, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=e2b61e4583cc)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640587, encodeId=6241164058e41, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=FAKTION研究: PI3K/AKT通路抑制剂带来新希望, objectType=article, longId=167060, objectId=e32e16e06047, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=e32e16e06047, replyNumber=0, likeNumber=65, createdTime=2019-12-24, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=e32e16e06047, moduleTitle=FAKTION研究: PI3K/AKT通路抑制剂带来新希望, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=e32e16e06047)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640586, encodeId=8a6c16405861c, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Int J Mol Sci：石墨烯诱导的成骨分化由整合素/ FAK轴介导, objectType=article, longId=159394, objectId=ff7215939420, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=ff7215939420, replyNumber=0, likeNumber=68, createdTime=2019-12-25, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=ff7215939420, moduleTitle=Int J Mol Sci：石墨烯诱导的成骨分化由整合素/ FAK轴介导, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=ff7215939420)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640584, encodeId=fce71640584e0, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Brit J Cancer：类血管生成素蛋白3能够阻断FAK的细胞核输入并有助于索拉非尼响应, objectType=article, longId=145659, objectId=d9b514565909, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=d9b514565909, replyNumber=0, likeNumber=85, createdTime=2018-09-24, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=d9b514565909, moduleTitle=Brit J Cancer：类血管生成素蛋白3能够阻断FAK的细胞核输入并有助于索拉非尼响应, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=d9b514565909)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640585, encodeId=ea3916405850d, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=ACS Appl Mater Interfaces：纳米纤维通过FAK/RhoA/YAP1通路调节单个骨髓干细胞成骨, objectType=article, longId=148438, objectId=016c148438a5, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=016c148438a5, replyNumber=0, likeNumber=64, createdTime=2018-10-20, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=016c148438a5, moduleTitle=ACS Appl Mater Interfaces：纳米纤维通过FAK/RhoA/YAP1通路调节单个骨髓干细胞成骨, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=016c148438a5)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640581, encodeId=8849164058191, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Cell Prolif：Fak-Mapk、Hippo和Wnt信号通路在牵张成骨中的表达和调节, objectType=article, longId=132636, objectId=7d781326362b, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=7d781326362b, replyNumber=0, likeNumber=0, createdTime=2018-12-13, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=7d781326362b, moduleTitle=Cell Prolif：Fak-Mapk、Hippo和Wnt信号通路在牵张成骨中的表达和调节, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=7d781326362b)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640583, encodeId=6de91640583ab, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Biomed Pharmacother：FAK和BMP-9通过增强Wnt-β-连环蛋白信号传导协同触发脂肪来源干细胞的成骨分化和骨形成, objectType=article, longId=141162, objectId=7f901411622f, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=7f901411622f, replyNumber=0, likeNumber=63, createdTime=2018-12-24, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=7f901411622f, moduleTitle=Biomed Pharmacother：FAK和BMP-9通过增强Wnt-β-连环蛋白信号传导协同触发脂肪来源干细胞的成骨分化和骨形成, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=7f901411622f)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640582, encodeId=8a9d1640582eb, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Diabetologia：FAK酪氨酸磷酸化受AMPK调节并控制人骨骼肌的代谢, objectType=article, longId=136355, objectId=25f513635561, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=25f513635561, replyNumber=0, likeNumber=65, createdTime=2018-11-04, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=25f513635561, moduleTitle=Diabetologia：FAK酪氨酸磷酸化受AMPK调节并控制人骨骼肌的代谢, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=25f513635561)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640580, encodeId=31a1164058084, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Sci Signal：FAK导致肿瘤免疫逃逸的机制, objectType=article, longId=123042, objectId=7c2112304246, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=7c2112304246, replyNumber=0, likeNumber=76, createdTime=2018-07-01, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=7c2112304246, moduleTitle=Sci Signal：FAK导致肿瘤免疫逃逸的机制, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=7c2112304246)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640579, encodeId=89bb16405e909, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=ANN ONCOL：FAK蛋白表达与结直肠癌抗血管生成治疗反应有关, objectType=article, longId=118275, objectId=827d1182e556, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=827d1182e556, replyNumber=0, likeNumber=65, createdTime=2018-09-13, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=827d1182e556, moduleTitle=ANN ONCOL：FAK蛋白表达与结直肠癌抗血管生成治疗反应有关, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=827d1182e556)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640578, encodeId=292216405e863, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Oncogene：PDE4D促进BRAF突变黑色素瘤中FAK介导的细胞侵袭, objectType=article, longId=88059, objectId=613888059d5, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=613888059d5, replyNumber=0, likeNumber=75, createdTime=2017-05-06, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=613888059d5, moduleTitle=Oncogene：PDE4D促进BRAF突变黑色素瘤中FAK介导的细胞侵袭, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=613888059d5)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640577, encodeId=8f9016405e780, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=JCB：FAK帮助肿瘤细胞越狱, objectType=article, longId=29834, objectId=7a1129834f9, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=7a1129834f9, replyNumber=0, likeNumber=66, createdTime=2014-11-15, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=7a1129834f9, moduleTitle=JCB：FAK帮助肿瘤细胞越狱, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=7a1129834f9)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640575, encodeId=c58616405e584, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=Mol Cancer Ther：研究证实小分子药物CFAK-Y15可有效治疗脑癌, objectType=article, longId=18354, objectId=355d183543c, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=355d183543c, replyNumber=0, likeNumber=79, createdTime=2013-08-27, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=355d183543c, moduleTitle=Mol Cancer Ther：研究证实小分子药物CFAK-Y15可有效治疗脑癌, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=355d183543c)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640576, encodeId=d2a416405e6a1, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=NAT COMMUN：对将FAK抑制因子作为抗癌药物开发的再思考, objectType=article, longId=20804, objectId=11ba208045b, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=11ba208045b, replyNumber=0, likeNumber=60, createdTime=2013-09-09, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=11ba208045b, moduleTitle=NAT COMMUN：对将FAK抑制因子作为抗癌药物开发的再思考, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=11ba208045b)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640574, encodeId=0b2616405e4a6, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=MOL CANCER THER:CFAK-Y15可治疗神经胶质瘤, objectType=article, longId=13711, objectId=43a513e1167, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=43a513e1167, replyNumber=0, likeNumber=62, createdTime=2013-03-10, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=43a513e1167, moduleTitle=MOL CANCER THER:CFAK-Y15可治疗神经胶质瘤, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=43a513e1167)], followStatus=false, userIsMember=false, type=1, lengthMark=0), GetTopicListResponse(id=1640573, encodeId=a4da16405e301, content=<a href='/topic/show?id=78a4e22688' target=_blank style='color:#2F92EE;'>#FAK#</a>, objectTitle=PLoS ONE：microRNA-7降低FAK表达抑制乳腺癌转移, objectType=article, longId=8919, objectId=4ce0891929, topicUrl=null, isHasObj=0, objectCover=null, objectUrl=/article/show_article.do?id=4ce0891929, replyNumber=0, likeNumber=75, createdTime=2012-09-09, rootId=0, userName=12498a49m67暂无昵称, userId=1a7722667526, projectId=1, avatar=, status=1, hasArticle=1, attachment=null, ipAttribution=, moduleDTOList=[ModuleDTO(moduleId=4ce0891929, moduleTitle=PLoS ONE：microRNA-7降低FAK表达抑制乳腺癌转移, moduleType=article, hrefUrl=https://www.medsci.cn/article/show_article.do?id=4ce0891929)], followStatus=false, userIsMember=false, type=1, lengthMark=0)] 梅斯话题小助手

FAK

2020-05-29

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

ASCO 2022 | FAKTION临床：capivasertib+氟维司群，改善芳香化酶抑制剂耐药MBC患者生存！

2022-06-14

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Diabetologia：IGFBP-1在人2型糖尿病肾小球中的表达降低并调节人足细胞中的β1-整联蛋白/FAK信号传导

2021-11-21

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Artif Cells Nanomed Biotechnol：FAK在滑膜间充质干细胞中通过Wnt和MAPK信号通路介导BMP9诱导的成骨分化

2020-06-07

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

FAKTION研究: PI3K/AKT通路抑制剂带来新希望

2019-12-24

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Int J Mol Sci：石墨烯诱导的成骨分化由整合素/ FAK轴介导

2019-12-25

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Brit J Cancer：类血管生成素蛋白3能够阻断FAK的细胞核输入并有助于索拉非尼响应

2018-09-24

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

ACS Appl Mater Interfaces：纳米纤维通过FAK/RhoA/YAP1通路调节单个骨髓干细胞成骨

2018-10-20

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Cell Prolif：Fak-Mapk、Hippo和Wnt信号通路在牵张成骨中的表达和调节

2018-12-13

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Biomed Pharmacother：FAK和BMP-9通过增强Wnt-β-连环蛋白信号传导协同触发脂肪来源干细胞的成骨分化和骨形成

2018-12-24

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Diabetologia：FAK酪氨酸磷酸化受AMPK调节并控制人骨骼肌的代谢

2018-11-04

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Sci Signal：FAK导致肿瘤免疫逃逸的机制

2018-07-01

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

ANN ONCOL：FAK蛋白表达与结直肠癌抗血管生成治疗反应有关

2018-09-13

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Oncogene：PDE4D促进BRAF突变黑色素瘤中FAK介导的细胞侵袭

2017-05-06

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

JCB：FAK帮助肿瘤细胞越狱

2014-11-15

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

Mol Cancer Ther：研究证实小分子药物CFAK-Y15可有效治疗脑癌

2013-08-27

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

NAT COMMUN：对将FAK抑制因子作为抗癌药物开发的再思考

2013-09-09

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

MOL CANCER THER:CFAK-Y15可治疗神经胶质瘤

2013-03-10

12498a49m67暂无昵称 (审核中...)

前往app查看评论内容

#FAK#

PLoS ONE：microRNA-7降低FAK表达抑制乳腺癌转移

2012-09-09

科室
- - 订阅+
  - 更多科室
工具
服务